JM Littleton

First name:
JM
Last name:
Littleton
Prendergast, M. A., Rogers, D. T., Mulholland, P. J., Littleton, J. M., LH, W., Self, R. L., & Nath, A. (2002). Neurotoxic effects of the human immunodeficiency virus type-1 transcription factor Tat require function of a polyamine sensitive-site on the N-methyl-D-aspartate receptor. Brain Research, 954(2), 300-7. https://doi.org/10.1016/s0006-8993(02)03360-7 (Original work published 2002)
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Mulholland, P. J., Self, R. L., Harris, B. R., Littleton, J. M., & Prendergast, M. A. (-)-nicotine ameliorates corticosterone's potentiation of N-methyl-d-aspartate receptor-mediated cornu ammonis 1 toxicity. Neuroscience, 125(3), 671-82. https://doi.org/10.1016/j.neuroscience.2004.02.007 (Original work published 2004)
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Prendergast, M. A., Harris, B. R., Mullholland, P. J., JA, 2nd B., Gibson, D. A., Holley, R. C., & Littleton, J. M. Hippocampal CA1 region neurodegeneration produced by ethanol withdrawal requires activation of intrinsic polysynaptic hippocampal pathways and function of N-methyl-D-aspartate receptors. Neuroscience, 124(4), 869-77. https://doi.org/10.1016/j.neuroscience.2003.12.013 (Original work published 2004)
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Harris, B. R., Gibson, D. A., Prendergast, M. A., Blanchard, J. A., Holley, R. C., Scotland, R. L., et al. (2003). The neurotoxicity induced by ethanol withdrawal in mature organotypic hippocampal slices might involve cross-talk between metabotropic glutamate type 5 receptors and N-methyl-D-aspartate receptors. Alcoholism, Clinical And Experimental Research, 27(11), 1724-35. https://doi.org/10.1097/01.ALC.0000093601.33119.E3
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Mulholland, P. J., Harris, B. R., Wilkins, L. H., Self, R. L., Blanchard, J. A., Holley, R. C., et al. (1969). Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression. Alcohol (Fayetteville, N.y.), 31(1-2), 1-10. https://doi.org/10.1016/j.alcohol.2003.09.001 (Original work published 1969)
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Gibson, D. A., Harris, B. R., Prendergast, M. A., JA, 2nd B., Holley, R. C., Pedigo, N. W., et al. (2003). Polyamines contribute to ethanol withdrawal-induced neurotoxicity in rat hippocampal slice cultures through interactions with the NMDA receptor. Alcoholism, Clinical And Experimental Research, 27(7), 1099-106. https://doi.org/10.1097/01.ALC.0000075824.10502.DD
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Prendergast, M. A., Rogers, D. T., Barron, S., Bardo, M. T., & Littleton, J. M. (2002). Ethanol and nicotine: a pharmacologic balancing act?. Alcoholism, Clinical And Experimental Research, 26(12), 1917-8. https://doi.org/10.1097/01.ALC.0000040846.27378.80
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Harris, B. R., Prendergast, M. A., Gibson, D. A., Rogers, D. T., Blanchard, J. A., Holley, R. C., et al. (2002). Acamprosate inhibits the binding and neurotoxic effects of trans-ACPD, suggesting a novel site of action at metabotropic glutamate receptors. Alcoholism, Clinical And Experimental Research, 26(12), 1779-93. https://doi.org/10.1097/01.ALC.0000042011.99580.98
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Kelley, Z. D., Rogers, D. T., Littleton, J. M., & Lynn, B. C. (2019). Microfluidic capillary zone electrophoresis mass spectrometry analysis of alkaloids in Lobelia cardinalis transgenic and mutant plant cell cultures. Electrophoresis, 40(22), 2921-2928. https://doi.org/10.1002/elps.201900220
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Prendergast, M. A., Harris, B. R., Mayer, S., & Littleton, J. M. (2000). Chronic, but not acute, nicotine exposure attenuates ethanol withdrawal-induced hippocampal damage in vitro. Alcoholism, Clinical And Experimental Research, 24(10), 1583-92.
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