Polyamines contribute to ethanol withdrawal-induced neurotoxicity in rat hippocampal slice cultures through interactions with the NMDA receptor. Alcoholism, Clinical And Experimental Research, 27(7), 1099-106. https://doi.org/10.1097/01.ALC.0000075824.10502.DD
. (2003). JM Littleton
First name:
JM
Last name:
Littleton
Opposing effects of ethanol and nicotine on hippocampal calbindin-D28k expression. Alcohol (Fayetteville, N.y.), 31(1-2), 1-10. https://doi.org/10.1016/j.alcohol.2003.09.001 (Original work published 1969)
. (1969). The neurotoxicity induced by ethanol withdrawal in mature organotypic hippocampal slices might involve cross-talk between metabotropic glutamate type 5 receptors and N-methyl-D-aspartate receptors. Alcoholism, Clinical And Experimental Research, 27(11), 1724-35. https://doi.org/10.1097/01.ALC.0000093601.33119.E3
. (2003). Hippocampal CA1 region neurodegeneration produced by ethanol withdrawal requires activation of intrinsic polysynaptic hippocampal pathways and function of N-methyl-D-aspartate receptors. Neuroscience, 124(4), 869-77. https://doi.org/10.1016/j.neuroscience.2003.12.013 (Original work published 2004)
. (-)-nicotine ameliorates corticosterone's potentiation of N-methyl-d-aspartate receptor-mediated cornu ammonis 1 toxicity. Neuroscience, 125(3), 671-82. https://doi.org/10.1016/j.neuroscience.2004.02.007 (Original work published 2004)
. Selective vulnerability of hippocampal cornu ammonis 1 pyramidal cells to excitotoxic insult is associated with the expression of polyamine-sensitive N-methyl-D-asparate-type glutamate receptors. Neuroscience, 165(2), 525-34. https://doi.org/10.1016/j.neuroscience.2009.10.018 (Original work published 2010)
. (2010). Corticosterone increases damage and cytosolic calcium accumulation associated with ethanol withdrawal in rat hippocampal slice cultures. Alcoholism, Clinical And Experimental Research, 29(5), 871-81. https://doi.org/10.1097/01.alc.0000163509.27577.da
. (2005). Thiamine deficiency in the pathogenesis of chronic ethanol-associated cerebellar damage in vitro. Neuroscience, 135(4), 1129-39. https://doi.org/10.1016/j.neuroscience.2005.06.077 (Original work published 2005)
. Corticosterone and dexamethasone potentiate cytotoxicity associated with oxygen-glucose deprivation in organotypic cerebellar slice cultures. Neuroscience, 136(1), 259-67. https://doi.org/10.1016/j.neuroscience.2005.07.043 (Original work published 2005)
. A 24 h corticosterone exposure exacerbates excitotoxic insult in rat hippocampal slice cultures independently of glucocorticoid receptor activation or protein synthesis. Brain Research, 1082(1), 165-72. https://doi.org/10.1016/j.brainres.2006.01.069 (Original work published 2006)
. (2006).